Role of Zika virus on neuron development (foetal).

The Zika Virus Capsid Disrupts Corticogenesis by Suppressing Dicer Activity and miRNA Biogenesis Neural progenitor cell pyroptosis contributes to Zika virus-induced brain atrophy and represents a therapeutic target Upregulation of MicroRNA miR-9 Is Associated with Microcephaly and Zika Virus Infection in Mice Zika virus alters DNA methylation status of genes involved in Hippo signaling pathway in human neural progenitor cells Zika virus differentially infects human neural progenitor cells according to their state of differentiation and dysregulates neurogenesis through the Notch pathway Genome-wide Integrative Analysis of Zika-Virus-Infected Neuronal Stem Cells Reveals Roles for MicroRNAs in Cell Cycle and Stemness Zika virus induces astrocyte differentiation in neural stem cells Proteomic analysis of Zika virus infected primary human fetal neural progenitors suggests a role for doublecortin in the pathological consequences of infection in the cortex Zika Virus Alters DNA Methylation of Neural Genes in an Organoid Model of the Developing Human Brain Strain-Dependent Consequences of Zika Virus Infection and Differential Impact on Neural Development Zika virus E protein alters the properties of human fetal neural stem cells by modulating microRNA circuitry Zika-Virus-Encoded NS2A Disrupts Mammalian Cortical Neurogenesis by Degrading Adherens Junction Proteins Zika virus dysregulates gene expression and alters protein secretion in neural stem cells MicroRNA and mRNA dysregulation in astrocytes infected with Zika virus Recent Zika Virus Isolates Induce Premature Differentiation of Neural Progenitors in Human Brain Organoids Zika virus infection dysregulates human neural stem cell growth and inhibits differentiation into neuroprogenitor cells Zika Virus NS4A and NS4B Proteins Deregulate Akt-mTOR Signaling in Human Fetal Neural Stem Cells to Inhibit Neurogenesis and Induce Autophagy Zika Virus Disrupts Neural Progenitor Development and Leads to Microcephaly in Mice Extra Zika virus infected primary microglia impairs NPCs proliferation and differentiation Zika virus infection leads to mitochondrial failure, oxidative stress and DNA damage in human iPSC-derived astrocytes Zika virus infected primary microglia impairs NPCs proliferation and differentiation